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Tation. Repeated experiments showed consistency of our RT-PCR analysis.Statistical evaluation Results are expressed as percentages or medians and ranges. Statistical analysis included two sided Student's ttest reported as mean standard error. Statistical significance was considered at p
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L. J Cancer Res Clin Oncol 2011, 137(5):897?05. 29. Ellis MJ, Tao Y, Luo J, A'Hern R, Evans DB, Bhatnagar AS, Chaudri Ross HA, von Kameke A, Miller WR, Smith I, et al: Outcome prediction for estrogen receptor-positive breast cancer based on postneoadjuvant endocrine therapy tumor characteristics. J Natl Cancer Inst 2008, 100(19):1380?388. 30. Dowsett M, Smith IE, Ebbs SR, Dixon JM, Skene A, A'Hern
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Epartment of Veterans Affairs Merit Review Award, and by funds from Belluck and Fox, LLP New York, New York. We thank the NYU Cancer Institute Genomics Facility for expert assistance with gene expression profiling. 18.19.Gastrointestinal stromal tumors (GISTs) are the most common mesenchymal tumors of the digestive tract. GIST pathogenesis is most frequently attributed to gain-offunction mut
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N RBA-1 cells. Next, to determine whether activation of CaMKII by LTA was mediated through Ca2+ signaling, an upstream signaling molecule of CaM/CaMKII cascade, cells wereWang et al. Journal of Neuroinflammation 2010, 7:84 http://www.jneuroinflammation.com/content/7/1/Page 9 ofFigure 4 Involvement CaMKII in LTA-induced MMP-9 expression in RBA-1. (A, B) Cells were pretreated with inhibitors of calm
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N RBA-1 cells. Next, to determine whether activation of CaMKII by LTA was mediated through Ca2+ signaling, an upstream signaling molecule of CaM/CaMKII cascade, cells wereWang et al. Journal of Neuroinflammation 2010, 7:84 http://www.jneuroinflammation.com/content/7/1/Page 9 ofFigure 4 Involvement CaMKII in LTA-induced MMP-9 expression in RBA-1. (A, B) Cells were pretreated with inhibitors of calm